Chemoreception [electronic resource] : from cellular signaling to functional plasticity
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Author: International Symposium on Arterial Chemoreception (15th : 2002 : Lyon, France), Pequignot, Jean-Marc, Gonzalez, Constancio, Nurse, Colin A, Prabhakar, Nanduri R, Dalmaz, Yvette
Added by: sketch
Added Date: 2015-12-30
Language: eng
Subjects: Chemoreceptors, Arteries, Arteries, Chemoreceptors
Publishers: Boston, MA : Springer US
Collections: folkscanomy miscellaneous, folkscanomy, additional collections
ISBN Number: 9781441992802, 1441992804, 1461348730, 9781461348733
Pages Count: 600
PPI Count: 600
PDF Count: 1
Total Size: 314.04 MB
PDF Size: 58.53 MB
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Total Files: 18
Media Type: texts
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Chemoreception: From Cellular Signaling to Functional Plasticity
Author: Jean-Marc Pequignot, Constancio Gonzalez, Colin A. Nurse, Nanduri R. Prabhakar, Yvette Dalmaz
Published by Springer US
ISBN: 978-1-4613-4873-3
DOI: 10.1007/978-1-4419-9280-2
Table of Contents:
The volume presents up to date major aspects of the chemoreceptor pathway from molecular and cellular signalling processes in the carotid body and other oxygen-sensing structures to central integration of chemosensory inputs and systemic implications. Because sustained changes in oxygen homeostasis are able to induce morphofunctional remodeling which remains poorly understood but may provide further ways of research in physiology and pathophysiology, two sessions were devoted to the mechanisms involved in the processes of plasticity induced by long-term or intermittent hypoxia during the perinatal period or at adulthood
Author: Jean-Marc Pequignot, Constancio Gonzalez, Colin A. Nurse, Nanduri R. Prabhakar, Yvette Dalmaz
Published by Springer US
ISBN: 978-1-4613-4873-3
DOI: 10.1007/978-1-4419-9280-2
Table of Contents:
- Honoring Pierre Dejours : His Contribution to the Study of the Role of the Arterial Chemoreceptors in the Regulation of Breathing in Humans
- 2P Domain K+ Channels: Novel Pharmacological Targets for Volatile General Anesthetics
- Ca2+ responses to hypoxia are mediated by IP3-R on Ca2+ store depletion
- Functional Identification of Kvα Subunits Contributing to the O2-Sensitive K+ Current in Rabbit Carotid Body Chemoreceptor Cells
- Carotid Body Chemoreceptor Activity in Mice Deficient in Selected Subunits of NADPH Oxidase
- Glucose Sensing Cells in the Carotid Body
- Effect of Mitochondrial Inhibitors on Type I Cells
- Ascorbate in the Carotid Body
- Studies on Glomus Cell Sensitivity to Hypoxia in Carotid Body Slices
- An Unusual Cytochrome a592 with Low PO2 Affinity correlates with Afferent Discharge in the Carotid Body
- A Reevaluation of the Mechanisms Involved in the Secretion of Catecholamine Evoked by 2, 4-Dinitro Phenol from Chemoreceptor Cells of the Rabbit Carotid Body
- Enhancing Effect of Vasopressin on the Hyperglycemic Response to Carotid Body Chemoreceptor Stimulation
- Immunohistochemical Study of the Carotid Body During Acute Hypoxia
- Effect of HERG-like Potassium Channel Blocker on the Carotid Body Chemoreception
- The Effect of Methanandamide on Isolated Type I Cells
- Doxapram Stimulates Carotid Body with Different Mechanisms from Hypoxic Chemotransduction
- Neuromuscular Blocking Agents and Carotid Body Oxygen Sensing
- The Pulmonary Interstitium: an Introductory Review
- Regulation of K+ Currents by CO in Carotid Body type I Cells and Pulmonary Artery Smooth Muscle Cells
- Ionotropic Receptors in Pulmonary Neuroepithelial Bodies (NEB) and their Possible Role in Modulation of Hypoxia Signalling
The volume presents up to date major aspects of the chemoreceptor pathway from molecular and cellular signalling processes in the carotid body and other oxygen-sensing structures to central integration of chemosensory inputs and systemic implications. Because sustained changes in oxygen homeostasis are able to induce morphofunctional remodeling which remains poorly understood but may provide further ways of research in physiology and pathophysiology, two sessions were devoted to the mechanisms involved in the processes of plasticity induced by long-term or intermittent hypoxia during the perinatal period or at adulthood
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